The Dynamic Nature of the Facial Skeleton
Many people assume that once a person reaches their late teens or early twenties, their facial bone structure is set for life. However, research into the science of aging has revealed a more complex and fascinating reality. The entire facial skeleton is in a constant state of flux, governed by bone remodeling, which involves the dual processes of bone deposition and bone resorption. In youth, these processes are relatively balanced, but as we age, that balance shifts, leading to perceptible changes in facial contour.
Bone Remodeling: Deposition vs. Resorption
Bone remodeling is a lifelong process where mature bone tissue is removed from the skeleton (resorption) and new bone tissue is formed (deposition). This process is vital for maintaining a healthy and robust skeletal structure. While bones throughout the body participate in this cycle, the facial skeleton has unique patterns of remodeling. In the midface, particularly around the cheekbones (zygoma), bone density and volume are gradually lost over time. This resorption process means that instead of growing, the cheekbones tend to recede and flatten, particularly after the age of 35.
The Midface and the Aging Process
The midface is one of the most susceptible areas to age-related bone loss. This loss of projection in the cheekbones significantly contributes to the characteristic changes seen in an aging face. The once prominent, high-set cheekbones of youth may become flatter and less defined. This is compounded by changes in other facial bones, such as the enlargement of the eye sockets (orbital aperture) and the recession of the maxilla (upper jawbone), which further diminishes the underlying skeletal support for the overlying soft tissues. Understanding these skeletal shifts is crucial for grasping why the face changes so dramatically over the decades.
The Interplay of Bone and Soft Tissue Changes
The perception of prominent or defined cheekbones is a result of both the underlying bone structure and the soft tissues that drape over it. As we age, changes in both components contribute to the overall effect. The loss of bone density in the cheeks and jaw is accompanied by the shifting and shrinking of facial fat pads. These fat pads, which provide youthful volume and contour, tend to descend and thin out. When combined with the receding bone structure, this soft tissue movement can exacerbate the appearance of flattened cheeks and deeper folds, such as the nasolabial folds.
Youthful vs. Aging Facial Structure
| Feature | Youthful Appearance (Early Adulthood) | Aging Appearance (Later Adulthood) |
|---|---|---|
| Cheekbones | High, projected, well-defined | Flatter, receded, less prominent |
| Facial Shape | Often described as an 'upside-down triangle' or heart-shaped, with fullness in the cheeks and a tapered chin | Becomes more 'pyramidal' or square, with wider jowls and less midface volume |
| Eye Sockets | Smaller, with taut skin and fat pads supporting the orbital rim | Orbital aperture widens, creating a sunken or hollowed-out appearance under the eyes |
| Skin & Fat | Firm, elastic, and supported by robust underlying bone structure | Loses volume, becomes less elastic, and sags, revealing the loss of underlying bone support |
Factors Influencing Facial Bone Changes
While aging is a universal experience, the rate and extent of facial bone changes can vary significantly from person to person. Several factors play a role in this process:
- Genetics: An individual's genetic predisposition is a primary determinant of their natural bone structure and how it will change over time. Some people are born with stronger, denser facial bones that hold up better against age-related resorption.
- Hormonal Changes: In women, hormonal shifts, particularly during and after menopause, can accelerate bone density loss throughout the body, including the face.
- Lifestyle: Factors such as diet, exercise, smoking, and sun exposure can all impact bone health. A diet rich in calcium and vitamin D is essential for maintaining bone density, while smoking is known to hinder bone remodeling.
- Dental Health: The presence and health of one's teeth significantly influence jawbone structure. Tooth loss can accelerate bone resorption in the maxilla and mandible, further altering the facial profile.
Modern Interventions for Enhancing Cheekbones
In the past, understanding of facial aging focused almost exclusively on skin and soft tissue. Today, a more comprehensive approach recognizes the importance of the underlying bone structure. For individuals concerned about age-related changes to their cheekbones, several cosmetic options are available to restore a more youthful appearance:
- Dermal Fillers: Injectable fillers, such as hyaluronic acid, can be used to add volume and projection to the cheek area, effectively mimicking the lost bone and fat. These treatments are temporary but offer a non-surgical solution.
- Fat Grafting: This procedure involves harvesting fat from one area of the body and injecting it into the cheeks. Fat grafting provides a longer-lasting, more natural-feeling result than synthetic fillers.
- Facial Implants: For a permanent change, synthetic implants can be surgically placed to augment the cheekbones. This option is more invasive but offers a lasting solution for more significant structural changes.
For more information on bone health, consult resources from organizations like the National Institutes of Health.
Conclusion
The question of whether cheekbones continue to grow has a layered answer. While they stop growing in the adolescent sense, the facial skeleton, including the cheekbones, undergoes continuous remodeling. This process involves a gradual loss of bone density and projection in key areas like the cheeks, which is a major contributor to facial aging. The changes are not solely due to sagging skin but are a complex interplay of bone resorption and soft tissue redistribution. By understanding this intricate process, we can better appreciate the science behind facial aging and explore the most effective ways to address these changes.